Telomerase activation, mitochondrial signaling, and NAD+ metabolism
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Longevity research peptides target the fundamental molecular mechanisms of aging: telomere shortening, mitochondrial dysfunction, and NAD+ depletion. By addressing these three pillars simultaneously, researchers can investigate aging at the chromosomal, organelle, and enzymatic levels.
Epithalon activates hTERT expression to maintain telomere length. MOTS-c is a mitochondrial-derived peptide that activates AMPK and translocates to the nucleus under metabolic stress. NAD+ serves as a substrate for sirtuins, PARPs, and CD38, with age-related decline correlating to mitochondrial dysfunction and cellular senescence.
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